TY - JOUR
AU - 
AB - THE JOURNAL OF BIOLOGICAL CHEMISTRY VOL. 283, NO. 49, pp. 34029 –34036, December 5, 2008 © 2008 by The American Society for Biochemistry and Molecular Biology, Inc. Printed in the U.S.A. □ S Received for publication, April 18, 2008, and in revised form, September 16, 2008 Published, JBC Papers in Press, October 2, 2008, DOI 10.1074/jbc.M803012200 Kepeng Wang, Chihao Wang, Fang Xiao, Haixia Wang, and Zhenguo Wu From the Department of Biochemistry, Hong Kong University of Science & Technology, Clearwater Bay, Kowloon, Hong Kong, China Skeletal muscle satellite cell-derived myoblasts are mainly (7–10). When grown in culture, proliferating myoblasts do not responsible for postnatal muscle growth and injury-induced express myogenin. Expression of myogenin signals that myo- regeneration. However, the cellular signaling pathways that blasts have irreversibly withdrawn from the cell cycle and have control proliferation and differentiation of myoblasts remain initiated the differentiation program. Both MyoD and MEF2 poorly defined. Recently, we found that JAK1/STAT1/STAT3 are known to be involved in myogenin induction at the start of not only participate in myoblast proliferation but also actively differentiation (11–13). prevent them from premature differentiation. Unexpectedly, we Skeletal muscle satellite cells, which are a group of quiescent found that a related 
TI - JAK2/STAT2/STAT3 Are Required for Myogenic Differentiation
JF - Journal of Biological Chemistry
DO - 10.1074/jbc.m803012200
DA - 2008-12-01
UR - https://www.deepdyve.com/lp/unpaywall/jak2-stat2-stat3-are-required-for-myogenic-differentiation-5qQ8YG3DqG
DP - DeepDyve
ER -