TY - JOUR
AU - 
AB - THE JOURNAL OF BIOLOGICAL CHEMISTRY Vol. 277, No. 17, Issue of April 26, pp. 14884 –14893, 2002 © 2002 by The American Society for Biochemistry and Molecular Biology, Inc. Printed in U.S.A. Cross-talk between ERK and p38 MAPK Mediates Selective Suppression of Pro-inflammatory Cytokines by Transforming Growth Factor- Received for publication, December 9, 2001, and in revised form, February 4, 2002 Published, JBC Papers in Press, February 12, 2002, DOI 10.1074/jbc.M111718200 Yi Qun Xiao, Ken Malcolm, G. Scott Worthen, Shyra Gardai, William P. Schiemann, Valerie A. Fadok, Donna L. Bratton, and Peter M. Henson‡ From the Program in Cell Biology, Department of Pediatrics, National Jewish Medical and Research Center, Denver, Colorado 80206 Phagocytosis of apoptotic cells by macrophages re- CC chemokine MCP-1 (1, 2) either unchanged or slightly in- sults in the production of transforming growth factor- creased. We have previously reported that the uptake of apo- (TGF-
), which plays an important role in induction of ptotic cells by macrophages can be mediated by a phospha- an anti-inflammatory phenotype and resolution of in- tidylserine receptor and that triggering this receptor is respon- prevents flammation. In this study, we show that TGF- sible for the increased production of TGF-. The 
TI - Cross-talk between ERK and p38 MAPK Mediates Selective Suppression of Pro-inflammatory Cytokines by Transforming Growth Factor-β
JF - Journal of Biological Chemistry
DO - 10.1074/jbc.m111718200
DA - 2002-04-01
UR - https://www.deepdyve.com/lp/unpaywall/cross-talk-between-erk-and-p38-mapk-mediates-selective-suppression-of-B0Jhhmk1RC
DP - DeepDyve
ER -