TY - JOUR
AU - 
AB - Downloaded from genesdev.cshlp.org on September 19, 2021 - Published by Cold Spring Harbor Laboratory Press RESEARCH COMMUNICATION translocation uncovered the BCL9 gene, and caused Essential role of BCL9-2 in the BCL9 overexpression in the tumors (Willis et al. 1998). Recently legless, the Drosophila ortholog of BCL9, was switch between 
-catenin’s found to act as a segment polarity gene and to function in adhesive and transcriptional the Wnt/
-catenin pathway in cooperation with pygopus (Kramps et al. 2002; Thompson et al. 2002). The role of functions BCL9 proteins in vertebrate development and tumor pro- 1 1,3 gression had not been determined. Felix H. Brembeck, Thomas Schwarz-Romond, 2,4 1 Jeroen Bakkers, Sabine Wilhelm, Results and Discussion Matthias Hammerschmidt, and 1,5 Walter Birchmeier Vertebrate BCL9-2 proteins show an overall amino acid sequence identity of 60%, and 35% identity to the hu- Max Delbrueck Center for Molecular Medicine, 13092 Berlin, man proto-oncogene product BCL9 (Fig. 1A; see Gen- Germany; Max Planck Institute for Immunobiology, Bank AY296057 to AY296061; Willis et al. 1998). Up to 79108 Freiburg, Germany 90% sequence identity was found in seven short clusters of 20–30 amino acids, which are also conserved in legless -Catenin controls both cadherin-mediated cell adhe- from Drosophila 
TI - Essential role of BCL9-2 in the switch between β-catenin's adhesive and transcriptional functions
JF - Genes & Development
DO - 10.1101/gad.317604
DA - 2004-09-01
UR - https://www.deepdyve.com/lp/unpaywall/essential-role-of-bcl9-2-in-the-switch-between-catenin-s-adhesive-and-cyQRO2NtDU
DP - DeepDyve
ER -