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Mediation of Inflammation by the Polymorphonuclear Leukocyte as a Sequela of Immune Reactions

Mediation of Inflammation by the Polymorphonuclear Leukocyte as a Sequela of Immune Reactions T a b l e 1 Mediation of Inflammation by Representative Constituents of PMN-Leukocyte Lysosomes the Polymorphonuclear Enzymes Miscellaneous Leukocyte as a Sequela of Acid RNAse Cationic polypeptides and Acid DNAse proteins Acid phosphatase Mucopolysaccharides Immune Reactions Alkaline phosphatase Glycoproteins Beta-glucuronidase Endogenous pyrogen 5-nucleotidase Phagocytin Cathepsins A, B, C, D, E Slow reacting substance of by Lysozyme anaphylaxis (SRS-A)-? NADP-oxidase N o r t o n S. T a ic h m a n Kinin-forming enzymes Kinin-destroying enzymes A l t h o u g h a b u n d a n t n u m b e r s of polymorphonuclear Peroxidase Aryl sulfatase A and B (PMN) leukocytes can be seen in inflamed gingiva, we Phospholipase still have no clear idea about why they are there or Plasminogen activator what they are doing. Because these cells represent a (urokinase?) Collagenase vital defense mechanism against other forms of micro­ Vascular permeability- bial infection, we have assumed that they act in a simi­ promoting proteases lar capacity in the region of the gingival crevice. This Hyaluronidase hypothesis may sound reasonable; however, it has yet to be substantiated by direct supportive evidence. It is true that periodontal disease is http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png Journal of Periodontology Wiley

Mediation of Inflammation by the Polymorphonuclear Leukocyte as a Sequela of Immune Reactions

Taichman, Norton S.
Journal of Periodontology , Volume 41 (4) – Apr 1, 1970
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References (17)

Publisher
Wiley
Copyright
© American Academy of Periodontology
ISSN
0022-3492
eISSN
1943-3670
DOI
10.1902/jop.1970.41.4.228
Publisher site
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Abstract

T a b l e 1 Mediation of Inflammation by Representative Constituents of PMN-Leukocyte Lysosomes the Polymorphonuclear Enzymes Miscellaneous Leukocyte as a Sequela of Acid RNAse Cationic polypeptides and Acid DNAse proteins Acid phosphatase Mucopolysaccharides Immune Reactions Alkaline phosphatase Glycoproteins Beta-glucuronidase Endogenous pyrogen 5-nucleotidase Phagocytin Cathepsins A, B, C, D, E Slow reacting substance of by Lysozyme anaphylaxis (SRS-A)-? NADP-oxidase N o r t o n S. T a ic h m a n Kinin-forming enzymes Kinin-destroying enzymes A l t h o u g h a b u n d a n t n u m b e r s of polymorphonuclear Peroxidase Aryl sulfatase A and B (PMN) leukocytes can be seen in inflamed gingiva, we Phospholipase still have no clear idea about why they are there or Plasminogen activator what they are doing. Because these cells represent a (urokinase?) Collagenase vital defense mechanism against other forms of micro­ Vascular permeability- bial infection, we have assumed that they act in a simi­ promoting proteases lar capacity in the region of the gingival crevice. This Hyaluronidase hypothesis may sound reasonable; however, it has yet to be substantiated by direct supportive evidence. It is true that periodontal disease is

Journal

Journal of Periodontology – Wiley

Published: Apr 1, 1970

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